An AAA is a focal dilatation of an aorta, and aneurysm is considered if infrarenal aorta is >3 cm in diameter. AAAs are mostly asymptomatic and found incidentally, but if symptoms do occur, most often is about pain. Rupture of the AAA is a main complication. The incidence of AAA is higher in men, individuals older than 60 years and smokers. Development of the AAA is a multifactorial, systemic process due to alterations in aortal wall biology which results with progressive thinning and weakening of the aortic wall as well as increased aortic diameter. The main mechanism of the rupture is biomechanical wall stress. More than 90% of aneurysms originate infrarenal and are diagnosed by imaging modalities. Clinical AAA is divided into: asymptomatic, symptomatic and ruptured. The classic triad of symptoms is hypotension, acute pain and pulsatile abdominal mass. Mortality rate of rAAA is still very high and reaches 90%. Two main principles in management are open repair (OR) and EVAR. Management depends on aortic diameter, symptoms, rupture risk assessment and perioperative mortality evaluation. Benefits of EVAR compared with OR include reduced hospital stay, less blood loss and lower ischemia of visceral organs. Randomized trials didn’t show a significant difference in a perioperative mortality rate between those two methods in patients with rAAA. EVAR is associated with slightly lower perioperative mortality so as less complications and is suggested as a method of choice for hemodynamic stable patients that fulfill the anatomic criteria. Long-term surveillance is no difference between the two methods. Repair of rAAA using an OR or EVAR is similar to elective AAA repair with some modifications due to urgency of the repair. Complications of rAAA repair are similar to those appearing after elective repair, just there is a higher incidence of complications such as myocardial infarction, respiratory failure, bowel, pelvic and peripheral ischemia and acute kidney injury.