prikaz prve stranice dokumenta Hemodynamic changes in the posterior cerebral circulation triggered by insufficient sympathetic innervation – Cause of primary intracerebral hemorrhage?
Access restricted to students and staff of home institution
Scientific paper - Original scientific paper
Hemodynamic changes in the posterior cerebral circulation triggered by insufficient sympathetic innervation – Cause of primary intracerebral hemorrhage?
Medical Hypotheses, 76 (2011), 5; 668-669. https://doi.org/10.1016/j.mehy.2011.01.027
Dunatov, Siniša; Antončić, Igor; Bralić, Marina

Institutional repository: Repository of the University of Rijeka, Faculty of Medicine

Cite this document

Dunatov, S., Antončić, I. & Bralić, M. (2011). Hemodynamic changes in the posterior cerebral circulation triggered by insufficient sympathetic innervation – Cause of primary intracerebral hemorrhage?. Medical Hypotheses, 76. (5), 668-669. doi: 10.1016/j.mehy.2011.01.027

Dunatov, Siniša, et al. "Hemodynamic changes in the posterior cerebral circulation triggered by insufficient sympathetic innervation – Cause of primary intracerebral hemorrhage?." Medical Hypotheses, vol. 76, no. 5, 2011, pp. 668-669. https://doi.org/10.1016/j.mehy.2011.01.027

Dunatov, Siniša, Igor Antončić and Marina Bralić. "Hemodynamic changes in the posterior cerebral circulation triggered by insufficient sympathetic innervation – Cause of primary intracerebral hemorrhage?." Medical Hypotheses 76, no. 5 (2011): 668-669. https://doi.org/10.1016/j.mehy.2011.01.027

Dunatov, S., Antončić, I. and Bralić, M. (2011) 'Hemodynamic changes in the posterior cerebral circulation triggered by insufficient sympathetic innervation – Cause of primary intracerebral hemorrhage?', Medical Hypotheses, 76(5), pp. 668-669. doi: 10.1016/j.mehy.2011.01.027

Dunatov S, Antončić I, Bralić M. Hemodynamic changes in the posterior cerebral circulation triggered by insufficient sympathetic innervation – Cause of primary intracerebral hemorrhage?. Medical Hypotheses [Internet]. 2011 [cited 2024 November 09];76(5):668-669. doi: 10.1016/j.mehy.2011.01.027

S. Dunatov, I. Antončić and M. Bralić, "Hemodynamic changes in the posterior cerebral circulation triggered by insufficient sympathetic innervation – Cause of primary intracerebral hemorrhage?", Medical Hypotheses, vol. 76, no. 5, pp. 668-669, 2011. [Online]. Available at: https://urn.nsk.hr/urn:nbn:hr:184:505575. [Accessed: 09 November 2024]

Metadata
Title (english)Hemodynamic changes in the posterior cerebral circulation triggered by insufficient sympathetic innervation – Cause of primary intracerebral hemorrhage?
AuthorSiniša Dunatov
AuthorIgor Antončić
AuthorMarina Bralić
Author's institutionUniversity of Rijeka
Faculty of Medicine
(Department of Neurology)
Scientific / art field,
discipline and subdiscipline		BIOMEDICINE AND HEALTHCARE
Clinical Medical Sciences
Neurology
Abstract (english)
Primary intracerebral hemorrhage (ICH) is caused by hypertensive disease of small penetrating blood vessels in the basal ganglia, brain stem and cerebellum. Those regions are supplied by arteries of the so-called posterior brain circulation with insufficient sympathetic innervation. We propose the following hypothesis: due to insufficient sympathetic innervation hemodynamic changes occur in the vascular bed of the posterior brain circulation serving as a key factor for arterial rupture. If autoregulation is insufficient to maintain normal cerebral blood flow, in abrupt rise in the blood pressure, the amount of blood is rising causing higher static pressure, and according to Laplace's law higher pressure and larger radius leads to higher wall tension and subsequent rupture of arterial wall previously weakened by prolonged hypertension.
Keywords (english)
Languageenglish
Publication typeScientific paper - Original scientific paper
Publication statusPublished
Peer reviewPeer review - international
Publication versionPublished version
Journal titleMedical Hypotheses
Numberingvol. 76, no. 5, pp. 668-669
p-ISSN0306-9877
DOIhttps://doi.org/10.1016/j.mehy.2011.01.027
URN:NBNurn:nbn:hr:184:505575
Publication2011
Document URLhttps://linkinghub.elsevier.com/retrieve/pii/S0306987711000296
Type of resourceText
Access conditionsInstitutional access
Terms of useLicence In copyright icon
Created on2023-07-05 06:26:50