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Regulation of immune cell function and differentiation by the NKG2D receptor
Cellular and Molecular Life Sciences, 68 (2011), 21; 3519-3529. https://doi.org/10.1007/s00018-011-0797-0
Zafirova, Biljana; Wensveen, Felix M.; Gulin, Maja; Polić, Bojan

Institucijski repozitorij: Repozitorij Medicinskog fakulteta u Rijeci

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Zafirova, B., Wensveen, F. M., Gulin, M. i Polić, B. (2011). Regulation of immune cell function and differentiation by the NKG2D receptor. Cellular and Molecular Life Sciences, 68. (21), 3519-3529. doi: 10.1007/s00018-011-0797-0

Zafirova, Biljana, et al. "Regulation of immune cell function and differentiation by the NKG2D receptor." Cellular and Molecular Life Sciences, vol. 68, br. 21, 2011, str. 3519-3529. https://doi.org/10.1007/s00018-011-0797-0

Zafirova, Biljana, Felix M. Wensveen, Maja Gulin i Bojan Polić. "Regulation of immune cell function and differentiation by the NKG2D receptor." Cellular and Molecular Life Sciences 68, br. 21 (2011): 3519-3529. https://doi.org/10.1007/s00018-011-0797-0

Zafirova, B., et al. (2011) 'Regulation of immune cell function and differentiation by the NKG2D receptor', Cellular and Molecular Life Sciences, 68(21), str. 3519-3529. doi: 10.1007/s00018-011-0797-0

Zafirova B, Wensveen FM, Gulin M, Polić B. Regulation of immune cell function and differentiation by the NKG2D receptor. Cellular and Molecular Life Sciences [Internet]. 06.09.2011. [pristupljeno 23.05.2024.];68(21):3519-3529. doi: 10.1007/s00018-011-0797-0

B. Zafirova, F. M. Wensveen, M. Gulin i B. Polić, "Regulation of immune cell function and differentiation by the NKG2D receptor", Cellular and Molecular Life Sciences, vol. 68, br. 21, str. 3519-3529, Rujan 2011. [Online]. Dostupno na: https://urn.nsk.hr/urn:nbn:hr:184:805924. [Citirano: 23.05.2024.]

Za citiranje koristite ovu mrežnu adresu: https://urn.nsk.hr/urn:nbn:hr:184:805924
Podaci o radu
Naslov (engleski)Regulation of immune cell function and differentiation by the NKG2D receptor
AutorBiljana Zafirova
AutorFelix M. Wensveen
AutorMaja Gulin
AutorBojan Polić
Autorova ustanovaSveučilište u Rijeci
Medicinski fakultet
(Katedra za histologiju i embriologiju)
Znanstveno / umjetničko
područje, polje i grana		BIOMEDICINA I ZDRAVSTVO
Temeljne medicinske znanosti
Sažetak (engleski)
NKG2D is one of the most intensively studied immune receptors of the past decade. Its unique binding and signaling properties, expression pattern, and functions have been attracting much interest within the field due to its potent antiviral and anti-tumor properties. As an activating receptor, NKG2D is expressed on cells of the innate and adaptive immune system. It recognizes stress-induced MHC class I-like ligands and acts as a molecular sensor for cells jeopardized by viral infections or DNA damage. Although the activating functions of NKG2D have been well documented, recent analysis of NKG2D-deficient mice suggests that this receptor may have a regulatory role during NK cell development. In this review, we will revisit known aspects of NKG2D functions and present new insights in the proposed influence of this molecule on hematopoietic differentiation.
Ključne riječi (engleski)
Jezikengleski
Vrsta publikacijeZnanstveni rad - Izvorni znanstveni rad
Status objaveObjavljen
Vrsta recenzijeRecenziran - međunarodna recenzija
Verzija publikacijeObjavljena verzija rada (izdavačev PDF)
Naslov časopisaCellular and Molecular Life Sciences
Brojčani podacivol. 68, br. 21, str. 3519-3529
p-ISSN1420-682X
e-ISSN1420-9071
DOIhttps://doi.org/10.1007/s00018-011-0797-0
URN:NBNurn:nbn:hr:184:805924
Datum objave publikacije2011-09-06
Datum objave publikacije2011-11
ProjektŠifra: 062-0621261-1271
Naziv (hrvatski): Uloga NKG2D u razvoju, homeostazi i efektorskim funkcijama imunološkog sustava
Voditelj: Bojan Polić
Pravna nadležnost: Hrvatska
Financijer: MZOS
Linija financiranja: ZP
ProjektŠifra: 256686
Naziv (engleski): Upgrading the capacities for research in translational medicine at the Faculty of Medicine University of Rijeka
Kratica: TRANSMEDRI
Pravna nadležnost: eu
Financijer: EC
Linija financiranja: FP7
URL dokumentahttp://link.springer.com/10.1007/s00018-011-0797-0
Vrsta resursaTekst
Prava pristupaOtvoreni pristup
Uvjeti korištenjaIkona licence Imenovanje 4.0 međunarodna (CC BY 4.0)
Datum i vrijeme pohrane2018-11-30 07:32:46