Globally, abdominal aortic aneurysms (AAA) pose a significant health threat, with mortality rates among affected individuals surpassing those of the general population. Thus, early identification and intervention are essential for the effective therapy of these patients. AAA arises from aortic wall degeneration, resulting in irreversible dilation, exceeding normal diameter by over 50%. Predominantly affecting males, its occurrence ranges from 4 to 8 percent, with smoking as a key factor. AAA is multifactorial in origin, with genetic predisposition, advancing age, gender, smoking, and ethnicity. The pathogenesis of infrarenal aortic aneurysms involves factors such as tissue susceptibility, inflammation, and irreversible breakdown of ECM via proteolysis and apoptosis of VSCM in the aorta. Clinical presentation varies from asymptomatic to symptomatic, guiding treatment pathways. The gold standard diagnostic tools include ultrasound and CTA. Treatments encompass conservative measures, pharmacological intervention, and invasive options like endovascular repair (EVAR) or open surgery. Post-EVAR complications are characteristically endoleaks and stent migration, while groin hematoma, infection, dissection, pseudoaneurysm formation, bowel and spinal cord ischemia along with renal artery occlusion and limb thrombosis are additional possibilities. Specific complications of open surgery include wound complications, incisional hernia, and end-organ ischemia. In the case of ruptured AAA (rAAA), prompt assessment of hemodynamic stability and anatomical suitability is crucial for determining the optimal surgical approach. An emphasis on anatomical suitability guides the choice of surgical method. Overall, understanding the complexities of AAA management and promptly preventing the rupture of the aneurysm is critical for optimal patient outcomes.